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Targeting mitochondrial induces apoptosis Previous studies focusing on the regulation of mitochondria in apoptosis indicated that pro-apoptotic proteins translocate to the mitochondrial outer membrane and trigger the source of reactive oxygen species (ROS) and apoptosis directly

Advances in the use of hydroxyurea

In tendon and ligament pathology, both peptides have shown remarkable efficacy in preclinical models of tissue repair

However, Fe 3+ produced by the Fenton reaction is subsequently reduced to Fe 2+ by cellular reductase, creating a vicious cycle that further damages dopaminergic neurons

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