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These findings suggest that the OGC accounts for the low-affinity high capacity of GSH transport in liver mitochondria, and imply that the nature of the high affinity GSH transporter remains to be identified

[29] reported that H3K4me1 deposition is largely RORt-independent

This disruption promotes chemokine release in HIV-infected glial cells, which in turn recruits peripheral immune cells and induces a pro-inflammatory response within glial and peripheral immune cells [162,163,164]

Our data indicate that SLC25A39 functions as a tumor-promoting factor in LUAD progression

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