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Wilson's disease The protein, ATP7B, is responsible for the excretion of hepatic copper into the biliary tract, and its impairment in Wilson's disease results in an increased concentration of 'free' copper (i.e., not bound to the copper-carrying protein, ceruloplasmin) in blood, an increased excretion of copper in the urine (hypercupriuria), the deposition of copper in part of the cornea (forming Kayser-Fleischer rings), and the accumulation of copper in the liver and brain (106)

Mineral Blend: Includes magnesium, zinc, manganese, and copper and is intended for IM injection or IV infusion only

E.LeichtleA

Order: 1mg Purity: 98% Supply Ability: 20ton

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