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Furthermore, excessive accumulation of NADH in conjunction with reduced NAD + availability can inhibit glyceraldehyde-3-phosphate dehydrogenase (GAPDH) activity in the glycolytic pathway, leading to glucose shunting to the glycolytic branch pathway (Yan et al., 2016

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Tracks both protocol efficacy and excess GH exposure (elevated IGF-1 above age-adjusted ranges warrants dose adjustment) Fasting glucose and insulin (HOMA-IR): GH is counter-regulatory to insulin