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Glutathione pool oxidation results in the inhibition of respiration and oxidative phosphorylation (OXPHOS), proton leaks-dependent respiration, solute import, and nutrient oxidation and Krebs cycle ux (reviewed in [ 88 , 89 ])

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Concurrent activation of the ascorbateglutathione (AsA-GSH) cycle maintained redox equilibrium during early stress phases, though prolonged exposure imposed redox constraints