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Doris Wilflingseder initiated the MUI AnimalFree Research Cluster at the Medical University of Innsbruck (MUI)

In summary, SLC7A5 overexpression was able to reverse the protective effects of YTHDF1 knockdown, revealing the critical role of SLC7A5 in the YTHDF1-mediated cytoprotective mechanism

Mif in the cerebrospinal fluid is decreased during relapsing-remitting while increased in secondary progressive multiple sclerosis

The DMN is composed of interconnected midline structures, including the sgACC, ventromedial PFC (vmPFC), dorsomedial PFC, precuneus/PCC complex, and mesotemporal structures