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Calcitriol intervention significantly restores vitamin D receptor expression, enhances autophagy flux, stimulates mucin/antimicrobial peptide production, and inhibits NF-B-mediated inflammatory responses

Particular attention is given to interventions that enhance synaptic plasticity, activate intrinsic neuroprotective pathways, and promote functional network regeneration (Figure 2)

This precision approach eliminates the temptation to stack medicationsyour personalized single agent is more effective

Since H2AX is also phosphorylated during unperturbed S phase 31 , this drop may be caused by depleting the pool of S phase cells due to the G2 arrest

Improves Balance