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Although the purpose of this screen was to identify genes that were required for the elevated intestinal expression of gcs-1p::gfp in prdx-2 mutant animals, in the course of screening, 50 RNAi clones were noted that increased the expression of gcs-1p::gfp still further (see Additional file 1: Table S2)

Multiple lines of evidence have suggested the involvement of dysregulation in the excitatory and inhibitory neurotransmission in the pathophysiology of ASD