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Conversely, downregulation of FABP4 by apelin/apelin receptor (APLNR)-mediated Forkhead box O1 (FOXO1) suppression in ECs limits excessive FA transport and tissue accumulation, contributing to atheroprotecton against vascular diseases such as atherosclerosis [86,87,88]

As a result, an accumulation of Lipid-OOH occurs: since GSH is the reducing co-substrate of GPX4, able to regulate the redox state of the cell-forming Lipid-OH (Figure 2A), GSH depletion can negatively regulate GPX4, triggering ferroptosis [19]

10.1016/j.expneurol.2015.02.032 Exp

She had gained twelve pounds over two years without any change she could identify

Fair question