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Overexpression of ferroptosis suppressor protein 1 (FSP1) or cytosolic GPX4 stimulates the genesis of ferroptosis-resistant CD8 + T cells without compromising their function, while the depletion of ferroptosis sensitivity-promoting enzyme acyl-CoA synthetase long-chain family member 4 (ACSL4) protected CD8 + T cells from ferroptosis but impaired antitumor CD8 + T cell response ( Other studies have demonstrated that cancer cells that have undergone ferroptosis can release high mobility group Box 1 (HMGB1) in an autophagy-dependent manner (Yu et al., 2015

Peer review Peer review information Nature Reviews Nephrology thanks Benjamin Stewart, Ulf Panzer and the other, anonymous, reviewer(s) for their contribution to the peer review of this work

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Upon analyzing the constituents of tears in PD patients as well as in a mouse model, tears exhibited markedly elevated norepinephrine and -2-macroglobulin activity

Spatially restricted patterning cues provided by heparin-binding VEGF-A control blood vessel branching morphogenesis