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Mitochondrial metabolism of reactive oxygen species

These anomalies would only be transient, and a new steady state would rapidly be reached, with normalization of plasma calcium and phosphate in response to increased PTH secretion and the well-known inhibitory effect of this hormone on the tubular reabsorption of phosphate (trade-off hypothesis of Bricker and Slatopolsky) (19)

Mitochondrial base editing is now a reality with the use of a cytosine base editor (DdCBE), which combines a transcription activator-like effector (TALE) array with a double-stranded DNA-specific cytidine deaminase (DddA), to mediate targeted CG-to-TA editing [47, 48]