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Human data remain limited, and conclusions about clinical outcomes cannot be definitively established at this time

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In parallel, it regulates the thioredoxin antioxidant system through targets such as thioredoxin-1 (TXN) and thioredoxin reductase-1 (TXNRD1), which collectively maintain the intracellular redox environment ( The activity of Nrf2 is primarily controlled through its interaction with Kelch-like ECH-associated protein 1 (Keap1), a cytoplasmic repressor that facilitates the ubiquitin-dependent proteasomal degradation of Nrf2 ( Figure 2 )