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Improved regulation of CBF in response to both transient hyper- and hypotension subsequently protects cerebral microvasculature from hyperperfusive injury and damage as well as ensuring cerebral perfusion is able to match metabolic demand during periods of reduced driving pressures (i.e., orthostatic stress) (Claassen et al., 2021)

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Emerging role of substance and energy metabolism associated with neuroendocrine regulation in tumor cells

Cao, P., et al., Therapeutic approaches to modulating glutathione levels as a pharmacological strategy in Alzheimers disease