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1.5-3.0 g/day for one year) in individuals with diabetic peripheral neuropathy (3, 88)

Astrocyte-endothelial interaction disruption within the neurovascular unit exacerbates barrier dysfunction, enabling blood-derived neurotoxins to infiltrate the parenchyma and induce tissue damage

From there, the dose can continue increasing to 10 mg, 12.5 mg, and ultimately 15 mg if needed and tolerated

This susceptibility positions mitochondria as primary victims of oxidative stress, emphasizing the importance of mitochondrial integrity in cellular function

LIMITED-EDITION HOLIDAY GIFT SETS (US & Canada) The sets you see in the graphics