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LAG3 is an important inhibitory immune checkpoint that, by binding to MHC II molecules, suppresses T cell activation and proliferation ( + T cells into the tumor microenvironment, but also promotes T cell exhaustion by downregulating the secretion of key cytokines, such as IFN- ( Using the TIDE algorithm, our study predicted immunotherapy outcomes and found that patients in the high-risk group had higher overall TIDE and immune exclusion scores, with significantly lower response and benefit rates compared to the low-risk group

References 1 FASEB JournalBiological activities of thymosin beta-4 mapped to active peptide sequences, including the fragment marketed as TB-500

Ethics statement The studies involving humans were approved by WCG Clinical Services North America

DNA damage and cancer immunotherapy: A STING in the tale

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