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Consequently, designing therapies that promote the transformation of CAF subtypes from tumor-promoting to tumor-suppressive phenotypes has become an important direction for future research

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log 2 FC = 20.46), a Ca 2+ regulator affecting acute kidney injury [39, 40], ERLINs, which regulate IP 3 receptors, DNAJs regulating degradation, and ARFs controlling G-protein coupled receptors (GPCRs)