Disclosures: Peter Lymberopoulos: Nothing to Disclose, Raphael Kirou: Nothing to Disclose, Anwar Uddin: Nothing to Disclose, Aboud Kaliounji: Nothing to Disclose, Anjiya Shaikh: Nothing to Disclose, Madeleine Hines Salge: Nothing to Disclose, Patrick Boddie: Nothing to Disclose, Vinh Tran: Nothing to Disclose, Howard (Tzu-Hao) Lee: Nothing to Disclose, Kavish Patidar: Madrigal Pharmaceuticals: Medical Advisory Board, Fasiha Kanwal: Nothing to Disclose, Anji Wall: Nothing to Disclose, Abbas Rana: Nothing to Disclose, George Cholankeril: Nothing to Disclose 1425 FRAILTY AND SARCOPENIA PREDICT PERI-OPERATIVE OUTCOMES IN PATIENTS UNDERGOING LIVER TRANSPLANTATION: ANALYSIS OF NATIONAL INPATIENT SAMPLE 2016-2020 Aalam Sohal 1 Sheena Bhushan 1 Isha Kohl 2 Srinivasan Dasarathy 3 Kris Kowdley 4 , 1 Liver Institute Northwest, Seattle, WA, 2 Department of Public Health, Icahn School of Medicine, Mount Sinai, New York, NY, 3 Department of Gastroenterology, Hepatology, and Nutrition, Cleveland Clinic, Cleveland, OH, 4 Liver Institute Northwest Background: Frailty and sarcopenia contribute to adverse clinical outcomes in patients with cirrhosis awaiting and after liver transplantation (LT)
In this study, the effects of the investigational products were measured using D-Squame tape strip samples to quantify GSH and glutathione disulfide levels, offering valuable insights into the impact of glutathione amino acid precursors on skins endogenous antioxidant defense
in Jersey City where he held the positions of inspector general and judge advocate
A preferred position is one that does not interfere with hybridization, i.e., does not interfere with the hydrogen bonding interactions needed for base pairing
10.3748/wjg.v26.i20.2533 Summary Keywords stress-induced liver injury, differential gene expression analysis, mitochondria, cannabidiol, oxidative stress Citation Huang C, Liang H, Liang X, Liu Y, Wang J, Jiang H, Kou X, Chen J and Huang L (2025) The protective role of cannabidiol in stress-induced liver injury: modulating oxidative stress and mitochondrial damage
