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However, it is noteworthy that there is no significant correlation between serum and cerebrospinal fluid IGFs concentrations [69] and therefore, it seems that systemic IGFs are not a major source of IGFs for CNS

SelH, selenoprotein H

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These adaptive mechanisms result in glomerular hyperfiltration and increased glomerular capillary pressure and are associated with kidney injury and hypertension in the long run [150]

Together, these transcriptomic and functional data exclude a requirement for canonical MT1 or MT2 receptor signaling in melatonin-mediated regulation of ILC2 function