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Moreover, NRF2 target genes are also critical mediators to enhance the metabolism of pro-tumorigenic xenobiotics

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Additional studies have linked SLC25A39 to metal homeostasis and neuronal injury, and identified it as a genetic risk factor for Parkinsons disease 19 Moreover, SLC25A39 has been shown to play a crucial role in mitochondrial heme biosynthesis, with its silencing impairing iron incorporation into protoporphyrin IX in murine erythroleukemia cells 20,21

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