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Activation of the CaR-CSE/H2S pathway confers cardioprotection against ischemia-reperfusion injury

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Although the physiological function of ferroptosis is poorly understood, its implication in several human diseases such as tumorigenesis [15, 16], CVDs (e.g., atherosclerosis [17], ischemia-reperfusion injury (IRI) [18], cardiomyopathy [19, 20], and heart failure [21]) has been widely reported

Cell Stem Cell 26(2):161-171.e4