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This novel discovery underscores the critical role of Notch signaling in regulating hepatic function during sepsis and identifies potential therapeutic targets for preventing or treating sepsis-related liver dysfunction, particularly in patients with pre-existing NASH or cirrhosis

However, although targeting EGFRvIII is a rational strategy to combat GBM, phase II clinical trials with the EGFR receptor tyrosine kinase inhibitor erlotinib and phase III clinical trials with the EGFRvIII vaccine rindopepimut have failed to robustly increase patient overall survival, highlighting the immense plasticity of GBM cell populations, which dampens treatment efficacies and hinders tumor management [5, 8, 9]

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