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Excess glutamate accumulates extracellularly during stroke, causing excessive NMDAR activation and neuroexcitotoxicity, as well as inducing NMDAR-mediated iron uptake (Cheah et al., 2006), as BBB dysfunction during stroke allows iron-containing substances to enter the brain and accumulate in areas of ischaemic brain tissue prior to neurodegeneration (Helal, 2008

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