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L., Cottom, L

& Morrison, I

In the CPZ + 40 Hz condition, both APCCC1 + and PDGFR + cells were significantly increased compared with the CPZ control group (Fig

This action appears to result from a combination of mechanisms including: blocking Na+ channels (possibly the most important mechanism) enhancing GABA-mediated inhibition in the CNS (by increasing GABA levels by a variety of mechanisms) increase in K+ currents block of T-type Ca2+ channels blockade of excitatory NMDA receptor-mediated excitation NOTE: sodium valproate is the salt form of valproic acid

Finally, the blots were dried, scanned and band intensities were quantified with ImageJ