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In summary, current data support a model in which both LOXs and nonenzymatic Fenton chemistry contribute to lipid peroxidation during ferroptosis

Senescent cells are primed for ferroptosis Ferroptosis is an iron-dependent form of cell death promoted by the accumulation of oxidized lipid membranes

Graphic Abstract Access this article Similar content being viewed by others Data Availability No datasets were generated or analysed during the current study

While Esm1 + cells from the retina and embryonic intestine share expression of signature genes such as Esm1 , Flt4 (encoding the receptor tyrosine kinase VEGFR3) or Lama4 (laminin 4), the two populations show substantial differences in gene expression

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