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These results highlight the potential of AST to interrupt molecular triggers of neuronal degeneration in environmentally induced PD models ( in vivo experiments using MPTP-treated mice revealed that AST reversed behavioral deficits, preserved tyrosine hydroxylase (TH) expression, and reduced neuronal apoptosis, even in the presence of miR-7 knockdown

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Somatostatin-evoked Abeta catabolism in the brain: Mechanistic involvement of alpha-endosulfine-KATP channel pathway

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