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Thus, as far as mitochondrial oxidative stress homeostasis is concerned, mGSH is a vital factor maintaining mitochondrial function, controlling cell survival/death, and its depletion causes cell damage and facilitates cell death [57, 59]

Consequently, any agent that targets NOXs would be a promising therapeutic approach for cancer

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In univariable analysis [A], identifying as Hispanic was associated with significantly reduced odds of achieving 7% TBWL (OR 0.28, 95% CI 0.09-0.88, p=0.02) and less mean %TBWL (5.6% vs 9.4%, p 2m) from 14% (untreated cells) to 5% and 9%, respectively