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Strikingly, higher SFB levels, as observed in MyD88 (an adaptor for different innate immune receptors which recognize microbial stimuli)-deficient mice models, protect mice of a diabetic genotype from the development of the disease, showing that the microbiota can exert both inhibiting and promoting effects [10]

11 The dynamic balance between different T-cell subsets controls the formation of vulnerable and obstructive plaques during atherosclerotic development

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