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Above results are in accordance with the outcomes of antifatigue-targeted clinical studies

Erastin upregulated the expression of lncRNA GABPB1-AS1, inhibited the translation of GABPB1 and the expression of the peroxidase gene, leading to the accumulation of ROS and MDA, and promoting tumor cell death, suggesting that GABPB1-AS1 may be the key to Erastin-induced ferroptosis [136, 137] (Table 2)

In splenocytes from WT mice, pretreatment of MS (0.4 mM, Gpx inhibitor) for 48 h inhibited Con A-induced cytokines such as IL-2, IFN- and TNF- (Figure 7a)

Moreover, nitrosylation, like phosphorylation, functions as a signaling pathway and plays a major role in regulating several physiological and pathological processes

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