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This mechanism facilitates the spread of A from platelets into the bloodstream and eventually into the brain tissue, adding to the overall amyloid accumulation (131, 135)

Research hypotheses include: Lipolysis and Fatty-Acid Oxidation: Possible activation of adipocyte lipase or modulation of acetyl-CoA carboxylase activity

2.2 The coexistence of CKD and sarcopenia increases the risk of adverse outcomes The coexistence of CKD and sarcopenia not only accelerates disease progression but also significantly increases the risk of adverse clinical outcomes in patients, including cardiovascular events, hospitalization rates, and all-cause mortality