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This reaction was proposed to act as a protective mechanism of sickle cell disease (SCD) RBCs

Other contributors include faulty axonal transport, resulting in buildup of neurofilaments and synaptic breakdown (Taylor et al., 2016), and genetic mutationssuch as SOD1 (causing harmful protein accumulation), FUS (interfering with RNA handling), and TBK1 or OPTN (which impair the cell's waste disposal system known as autophagy

Structurally the simplest of the -amino acids, it is very unreactive when incorporated into proteins