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Additionally, a study by Liu et al (41) demonstrated that miR-196a promotes the development of non-small cell lung cancer by downregulating the expression of GPX3 and activating the JNK pathway, thereby enhancing the proliferation, differentiation, self-renewal ability and invasiveness of cancer stem cells (CSCs) (41)

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Studies of the FHIT loci genomic structure identified LINE-1, a human transposable element that is presumably involved in genomic deletion breakpoints associated with cancer (58,59)

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Researchers have investigated the primary anticancer mechanisms of SFN and determined the concentrations required for their activation, as outlined in Table 1 [90]