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doi: 10.1186/ar4012 39 HinzeCHSuzukiMKlein-GitelmanMPassoMHOlsonJSingerNGet al

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These verdicts characterize the regulation of sortilin shedding and identify a novel mechanism by which sortilin ectodomain shedding acts as a regulatory switch for delivery of BDNF to the secretory pathway or to the lysosome, thus modulating the bioavailability of endogenous BDNF (Fig

In this process, highly active NSPs including NE, CatG, and PR3 are closely associated with disease progression and lung function decline

As an example, GSTs protect cellular DNA against oxidative damage that can lead to an increase of DNA mutations or induce DNA damage promoting carcinogenesis 9