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The authors sequenced the entire genomes of A/J and four strains that lack an ahl4 phenotype, and found that the rs29358506 single nucleotide polymorphism (SNP), located in exon 3 of CS, is the only exon or splice site variant within the 5.5 megabases (Mb) candidate region that is unique to the A/J strain, indicating that CS is the gene responsible for ahl4

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This expression pattern supports a role for the CaSR in mediating terminal differentiation of the cells

Haskell, E

J Neurol Neurosurg Psychiatry 84, 13181325.Google Scholar Smith, PJ, Blumenthal, JA, Babyak, MA et al