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Due to inappropriate assessment models, inadequate understanding of nanobio-interactions, etc., current macrophage nanomedicines rarely achieve practical clinical translation [311], as evidenced by their limited commercialization

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Concurrently, mtROS stimulates lipid peroxidation, ultimately leading to disruption of mitochondrial membrane potential and mitochondrial calcium buffering capacity (36)

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Cell viability, glucose-stimulated insulin secretion, cellular adenosine triphosphate, reactive oxygen species (ROS) levels and Rho kinase (ROCK) activities were measured