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Then, the TCA cycle is operating to produce reducing compounds to maintain cellular redox balance 38

The disease is caused by a mutation in the ATP7B gene, which codes for a protein that facilitates the incorporation of copper into proteins (such as ceruloplasmin) and also the transportation of copper into vesicles that allow it to be secreted in bile.1 The critical effect of a mutation in ATP7B is diminished copper secretion into bile, which leads to excess copper accumulation in the hepatocyte

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Meanwhile, in vivo NIR fluorescence and photoacoustic imaging have been utilized to guide the PDT treatment synergistically

The application of L-asparaginase as a plasma Gln-depleting agent with a clear therapeutic effect in solid tumors warrants further investigation