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First entry, August 12, 2014: July 2013: 4.8kg (10.9 lbs.) at regular check-up

The absence of a significant signal in our analysis may reflect limited statistical power or differences in allele frequency across populations, rather than a definitive lack of involvement of TMCO1 in POAG pathogenesis in non-European individuals

Such buffering could for example be mediated by activation of the Nrf2-Keap1-ARE pathway, which has been shown to be increased after polyphenol supplementation which in turn induces the expression of phase II detoxifying enzymes, responsible for reducing endogenous toxic metabolites [65]

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