glutathione reductase in caco-2 cells differentiated Apoptosis and glutathione: beyond an antioxidant Involvement of glutathione peroxidases in

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During the acute phase of the disease, but also several months or years after successful antibiotic therapy, a significant proportion of patients develop symptoms of hypothalamic-pituitary insufficiency (62)

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However, DCs can also contribute to detrimental outcomes by producing pro-inflammatory mediators like TNF-, a factor implicated in renal atrophy, tubular apoptosis, and glomerular endothelial damage [21]

The absence of oxidative stress presumably prevented cell death under cystine or GSH depletion alone, suggesting that cysteine and GSH cooperate to mitigate oxidative stress