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Current research spans pharmacological neuroprotection, modulation of convergent pathological pathways (e.g., TDP-43 proteinopathy, neuroinflammation) common to both fALS and sALS (1214), and innovative biological approaches like stem cell and gene therapy (15, 16)

Additionally, autopsy findings revealed that GHRP-6 significantly reduced pathological increases in relative heart and lung weights, aligning these measures with values observed in healthy animals [1]

One carbon metabolism is a network of interrelated biochemical pathways that donate and regenerate one-carbon units, including the methyl group (reviewed [19,20])

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These findings highlight coordinated and mechanistically congruent alterations in specific cellular processes and sub-networks, laying the ground for follow-up mechanistic intervention and validation studies in PD model systems