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Oh M-H, et al

The pathophysiology of IC/BPS is poorly understood, and theories include chronic inflammation, autoimmune dysregulation, bacterial cystitis, urothelial dysfunction, deficiency of the glycosaminoglycan (GAG) barrier and urine cytotoxicity

Figure 7 Genome-wide cytosine methylation changes in an HD model were first observed in mouse striatal neurons expressing either pathogenic (STHdhQ111) or normal (STHdhQ7) polyQ domains

Thus, we propose QMP engineering as a strategy to ensure a long plasma half-life and enhanced therapeutic efficacy of albumin fusion proteins despite C-terminal cleavage

Learn more about this and which deficiencies are more likely to occur