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It plays an indispensable role in DNA synthesis, red blood cell formation, and neurological function ( Figure 1 Vitamin B12 deficiency, defined as ciculatory B12 level below 150200 pg/mL (borderline 200300 pg/mL) (Figure 2), including megaloblastic anemia and irreversible neurological damage ( Figure 2 For individuals at risk of B12 deficiency, treatment is often necessary to maintain adequate circulatory levels ( Thus, an optimized carrier system is crucial for enhancing the bioavailability of supplemental oral supplemental B12

Methyl B12 is the most promising treatment for patients with severe neurological symptoms, bypassing several steps in the absorption cycle and helping to relieve or completely reverse symptoms

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The effects of human GH and its lipolytic fragment (AOD9604) on lipid metabolism following chronic treatment in obese mice and beta(3)-AR knockout mice