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These benefits are attributed to its ability to modulate oxidative stress and inflammation [69]restore epigenetic balance, and enhance metabolic efficiency through improved lipid and glucose regulation

For example, the acetylation-deficient TP53 variant, TP53[3KR], lacks the ability to induce apoptosis and cell cycle arrest but retains its tumor suppression function akin to wild-type TP53

Mol Biol Rep 42:947962 Aller I, Rouhier N, Meyer AJ (2013) Development of roGFP2-derived redox probes for measurement of the glutathione redox potential in the cytosol of severely glutathione-deficient rml1 seedlings

Coupling of NH 2 -modified HS02-52G (25 nmol) on MyOne beads resulted in 66.4 g (4.1 nmol, 16.4% of starting aptamer amount, 820 pmol/mg bead) aptamer immobilization on the beads corresponding to 13.3 g aptamer/mg of the beads

BM and MS-L contributed to manuscript revision, read, and approved the submitted version