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Similarly, in an in vivo study using a 3-nitro propionate model to examine the effects of CBG on Huntingtons disease pathology, treatment with CBG significantly attenuated the upregulation of COX-2, iNOS, and pro-inflammatory cytokines such as TNF- and IL-6 (Figure 5) [107]

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Trx also functions as a protein disulfide isomerase that corrects disulfide bridges that are formed in error

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doi: 10.1046/j.1440-1746.2000.02199.x 132 LlovetJMKelleyRKVillanuevaASingalAGPikarskyERoayaieSet al