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Irisin was revealed to have neuroprotective effects on PD by activating the ERK, P38MAPK and AMPK pathways and then upregulating Nrf2, HO-1, and Bcl-2 independently of NF-B, reducing the release of cytokines and the NLRP3 inflammasome and Caspase-1 [87]

doi:10.1111/bcp.12362

Since most of the proteins coded by mtDNA are involved in ETC, mutations and deletions in mtDNA would likely disturb ETC and increase ROS formation, creating a vicious cycle further inflicting mitochondrial damage [46]