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Role of glucagon-like peptide-1 receptor agonists in pediatric obesity and metabolic dysfunction associated steatotic liver disease

It is also likely the SM-HLCs have alterations in metabolic gene expression with resultant changes of TCA cycle/glycolytic/mitochondrial intermediates that are geared towards high proliferation, high plasticity and dedifferentiation

Mechanistically, acetyl-CoA, a derivative of BCKAs, inhibits the interaction between PR domain-containing protein 16 (PRDM16) and peroxisome proliferator-activated receptor- (PPAR-) by acetylating the k915 site of PRDM16 to maintain WAT characteristics

Individuals with obesity demonstrate elevated levels of ROS and compromised antioxidant defenses, with the imbalance between oxidants and antioxidants closely correlated with obesity parameters, including BMI (149, 180183)

These interactions are still under investigation, and findings are based only on laboratory observations