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Torsein M, Lindberg A, Svensson C, Jensen SK, Berg C, Waller KP, et al

In the pathogenesis of ferroptosis, the accumulation of ROS not only directly damages cellular structures but also triggers dysregulation of downstream inflammatory and immune responses by activating multiple signaling pathways, such as nuclear factor-B (NF-B) and mitogen-activated protein kinase (MAPK) pathways (An et al

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This accumulation is largely prevented by the Cori cycle, in which gluconeogenic tissues (mainly liver and renal cortex) bear the energy cost of removing excess muscle lactate

10.2147/IJN.S359300 81 SunS.ShenJ.JiangJ.WangF.MinJ