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Our results provided evidence that inhibition of protein translation alone is sufficient to rescue both mTORC1 signaling and the ISR, suggesting its major role in the regulation of amino acid sensing pathways in HepG2 cells

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[53] [54] This is because it requires significant damage to the liver to impair bilirubin excretion, hence minor impairment (in the absence of biliary obstruction or Gilbert syndrome) would not lead to jaundice

Recent findings have shed light on the molecular mechanisms underlying the development of malignant brain tumors [16, 35]