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Ketogenic Diet

Biochemical and histological analyses were performed on the hippocampal and cortical tissue, assessing oxidative stress markers, antioxidant enzyme activity, and glial activation through spectrophotometric assays and immunohistochemistry

95 Gene deletion or the lack of unstable methyl groups in the form of methionine and choline undermines the ability to synthesize SAM, which leads to the development of steatosis and its rapid progress toward NASH

Furthermore, 3 patients required 2nd reLT and 1 patient 3rd reLT

The mechanisms likely involve immune modulation (microglia in ALS can adopt a neurodegenerative phenotype that might be restrained by microbial signals) and metabolic support (providing nutrients like nicotinamide or SCFAs to neurons and glia) [214, 218]