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Fustaino V, Presutti D, Colombo T, Cardinali B, Papoff G, Brandi R, et al

146 Mice with -cell-specific disruption of Sel1L-HRD1 develop early-onset diabetes and glucose intolerance, reflecting impaired insulin secretion and -cell dysfunction

However, its coadministration with LPS greatly augmented PDCE2 glutathionylation and associated cell death, suggesting cross-talk between reductase systems and PDC redox regulation