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Since GSH is not carried inside the cell, exogenously administered GSH is most likely to act by increasing plasma GSH levels reducing luminal oxidative stress and increasing NO bioavailability in patients with endothelial dysfunction (220)

Severe neonatal jaundice from acute hemolysis can also arise from a G6PD-deficient infants exposure to environmental oxidative compounds

Homocysteine, cysteine, and related metabolites in maternal and fetal plasma in preeclampsia

Roles of host mitochondria in the development of COVID-19 pathology: could mitochondria be a potential therapeutic target