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LT4 also reduced intrahepatic lipid content in an uncontrolled study in euthyroid subjects with non-alcoholic fatty liver disease and type 2 diabetes, suggesting a possible future role for LT4 in this population [45]

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Consistent with the glucose-deprivation phenotype, insulin treatment, a potent activator of glucose utilization, significantly increased GLUD1 protein levels without altering mRNA expression (Fig